EMAIL: [email protected]
ACTION on HEALING1. At the fibroblast level:
Active cell, mitochondrial system, rough and developed ergatoplasm, which secretes in the interstitial tissue tropocollagen, proelastin, structural glycoproteins and proteins type proeoglycane.These different substances serve to elaborate the conjunctive fibres , the elastic fibers and membranes.
It's on the mitochondrial level that the cytochrome oxydase absorbs the beam in an elective manner
and from this stimulates the metabolism of the fibroblast , which causes the elaboration of the colagen fibres.
Some action on the nucleol level is also possible since we notice an
increase in the cellular pool by physiological mitosis.
Biochemical studies confirm a sensible increase of the deshydrogenase and
lactinodeshydrogenase succinic acid in the epithelium bordering the
wound which tends to prove the fibroblastic action and the collagen synthesis.
2. On the vascular level
Vascular regeneration by stimulation of the endothelial cells and production of a
neovascularization, essential in the healing process.
Here, also, it is an action on the mitochondrial level which allows a good development of this phase.
Multiplication and structured growth of the neo-endothelium.
Active cell, mitochondrial system, rough and developed ergatoplasm, which secretes in the interstitial tissue tropocollagen, proelastin, structural glycoproteins and proteins type proeoglycane.These different substances serve to elaborate the conjunctive fibres , the elastic fibers and membranes.
It's on the mitochondrial level that the cytochrome oxydase absorbs the beam in an elective manner
and from this stimulates the metabolism of the fibroblast , which causes the elaboration of the colagen fibres.
Some action on the nucleol level is also possible since we notice an
increase in the cellular pool by physiological mitosis.
Biochemical studies confirm a sensible increase of the deshydrogenase and
lactinodeshydrogenase succinic acid in the epithelium bordering the
wound which tends to prove the fibroblastic action and the collagen synthesis.
2. On the vascular level
Vascular regeneration by stimulation of the endothelial cells and production of a
neovascularization, essential in the healing process.
Here, also, it is an action on the mitochondrial level which allows a good development of this phase.
Multiplication and structured growth of the neo-endothelium.
ACTION on PAIN1. On the free endings level
- by preferential stimulation action of laser emission on the fibers A alpha, A delta ,B beta against the C fibers , which are nociceptive
2. On the medullar level:
- by the diminution of the secretion of the P substance in the center of the
intermediate neuron in the substancia gelatinosa.
- by increasing of the secretion of the enkephalines, which blocks the receptive site of the P substance
- by starting the gate control system contolling pain messages by hormonal
feedback - unproven action -
3. On the lesional level
- by stopping formation of ECPGE prostaglandins which are vasodilatating
and hyperalgesiating substances synthetised and liberated locally in response to a lesion.
4. On the bioelectric level:
- the low frequencies, in the 5 to 15 Hz range, are responsible for the analgesic action,
ie optimal perturbation of a nociceptive transmission.
- the continuous beam, by quick repolarization of the menbranes of the injured cells,
makes disappear or decrease the painfull signal emission and then the pain intensity and form.
- these frequencies (CW and few Hz) are the same for use in topic or distal modes.
- by preferential stimulation action of laser emission on the fibers A alpha, A delta ,B beta against the C fibers , which are nociceptive
2. On the medullar level:
- by the diminution of the secretion of the P substance in the center of the
intermediate neuron in the substancia gelatinosa.
- by increasing of the secretion of the enkephalines, which blocks the receptive site of the P substance
- by starting the gate control system contolling pain messages by hormonal
feedback - unproven action -
3. On the lesional level
- by stopping formation of ECPGE prostaglandins which are vasodilatating
and hyperalgesiating substances synthetised and liberated locally in response to a lesion.
4. On the bioelectric level:
- the low frequencies, in the 5 to 15 Hz range, are responsible for the analgesic action,
ie optimal perturbation of a nociceptive transmission.
- the continuous beam, by quick repolarization of the menbranes of the injured cells,
makes disappear or decrease the painfull signal emission and then the pain intensity and form.
- these frequencies (CW and few Hz) are the same for use in topic or distal modes.
ACTION on INFLAMMATION
By itself, the mesenchyme possesses the reactionnal plasticity, the multiple potentiality which allows the evolution of an inflammatory environment
The athermic lasers are effective in this case:
1. On the vascular level:
-the increase of the capillary permeability caused by the photonic beam, decreases or inhibits
the oedema and also the diapedese ;
- also the blood flow is not slowed down and the hemodynamic factors, as hydrostatic pressure
and oncotico-colloido-plasmatic pressure; regulating inflammation, are largely alleviated.
- increase of the permeability of small lymphatic vessels
2. On the multiplying level:
- mobilisation and metamorphosis of the mesenchymator cells
- mobilisation and multiplication of the epithelioid and hystiocitary cells, which are stimulated
through the intra-mitochondrial metabolism
3. At the prostaglandin secretion level
- decrease of the PGE2 (pro-inflammatory) ; this action is comparable to
the one of the acetylsalicilic acid
4.On the prostacyclines level:
- decrease
By itself, the mesenchyme possesses the reactionnal plasticity, the multiple potentiality which allows the evolution of an inflammatory environment
The athermic lasers are effective in this case:
1. On the vascular level:
-the increase of the capillary permeability caused by the photonic beam, decreases or inhibits
the oedema and also the diapedese ;
- also the blood flow is not slowed down and the hemodynamic factors, as hydrostatic pressure
and oncotico-colloido-plasmatic pressure; regulating inflammation, are largely alleviated.
- increase of the permeability of small lymphatic vessels
2. On the multiplying level:
- mobilisation and metamorphosis of the mesenchymator cells
- mobilisation and multiplication of the epithelioid and hystiocitary cells, which are stimulated
through the intra-mitochondrial metabolism
3. At the prostaglandin secretion level
- decrease of the PGE2 (pro-inflammatory) ; this action is comparable to
the one of the acetylsalicilic acid
4.On the prostacyclines level:
- decrease
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